Osteopontin Deficiency Attenuates Atherosclerosis in Female Apolipoprotein E–Deficient Mice-Reference-Cited by-同舟云学术

Osteopontin Deficiency Attenuates Atherosclerosis in Female Apolipoprotein E–Deficient Mice

Published:2003-06 Issue:6 Volume:23 Page:1029-1034
ISSN:1079-5642
Container-title:Arteriosclerosis, Thrombosis, and Vascular Biology
language:en
Short-container-title:ATVB

Author:

Matsui Yutaka¹,Rittling Susan R.¹,Okamoto Hiroshi¹,Inobe Manabu¹,Jia Nan¹,Shimizu Toshihiro¹,Akino Masatoshi¹,Sugawara Takeshi¹,Morimoto Junko¹,Kimura Chiemi¹,Kon Shigeyuki¹,Denhardt David¹,Kitabatake Akira¹,Uede Toshimitsu¹

Affiliation:

1. From the Division of Molecular Immunology (Y.M., M.I., J.M., C.K., S.K., T.U.), Institute for Genetic Medicine, Hokkaido University, Sapporo, Japan; Department of Cardiovascular Medicine (Y.M., H.O., N.J., T.S., M.A., T.S., A.K.), Hokkaido University Graduate School of Medicine, Sapporo, Japan; and Departments of Genetics (S.R.R.) and Cell Biology and Neuroscience (D.D.), Rutgers University, Piscataway, NJ.

Publisher

Ovid Technologies (Wolters Kluwer Health)

Subject

Cardiology and Cardiovascular Medicine

Reference31 articles.

1. High expression of genes for calcification-regulating proteins in human atherosclerotic plaques.

2. Osteopontin is elevated during neointima formation in rat arteries and is a novel component of human atherosclerotic plaques.

3. Osteopontin mRNA is expressed by smooth muscle-derived foam cells in human atherosclerotic lesions of the aorta.

4. Quantitation of atherosclerosis in murine models: correlation between lesions in the aortic origin and in the entire aorta, and differences in the extent of lesions between sexes in LDL receptor-deficient and apolipoprotein E-deficient mice

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