Folic Acid Treatment Reduces Chemokine Release From Peripheral Blood Mononuclear Cells in Hyperhomocysteinemic Subjects-Reference-Cited by-同舟云学术

Folic Acid Treatment Reduces Chemokine Release From Peripheral Blood Mononuclear Cells in Hyperhomocysteinemic Subjects

Published:2002-04 Issue:4 Volume:22 Page:699-703
ISSN:1079-5642
Container-title:Arteriosclerosis, Thrombosis, and Vascular Biology
language:en
Short-container-title:ATVB

Author:

Holven Kirsten B.¹,Aukrust Pål¹,Holm Torbjørn¹,Ose Leiv¹,Nenseter Marit S.¹

Affiliation:

1. From the Lipid Clinic (K.B.H., L.O., M.S.N.), MSD Cardiovascular Research Center (K.B.H., M.S.N.), Research Institute for Internal Medicine (K.B.H., P.A., T.H., M.S.N.), Section of Clinical Immunology and Infection Diseases, Medical Department (P.A.), and Department of Cardiology, Division of Heart and Lung Diseases (T.H.), Rikshospitalet University Hospital, Oslo, Norway.

Publisher

Ovid Technologies (Wolters Kluwer Health)

Subject

Cardiology and Cardiovascular Medicine

Reference29 articles.

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2. Ueland PM Refsum H Brattstrøm L. Plasma homocysteine and cardiovascular disease.In: Francis RBJ ed. Atherosclerotic Cardiovascular Disease Hemostasis and Endothelial Function. New York NY: Marcel Dekker; 1992: 182–222.

3. A Quantitative Assessment of Plasma Homocysteine as a Risk Factor for Vascular Disease

4. Endothelial cell injury due to copper-catalyzed hydrogen peroxide generation from homocysteine.

5. Effect of folic acid treatment on endothelium-dependent vasodilation and nitric oxide–derived end products in hyperhomocysteinemic subjects

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