Blockade of Gap Junctions In Vivo Provides Neuroprotection After Perinatal Global Ischemia-Reference-Cited by-同舟云学术

Blockade of Gap Junctions In Vivo Provides Neuroprotection After Perinatal Global Ischemia

Published:2005-10 Issue:10 Volume:36 Page:2232-2237
ISSN:0039-2499
Container-title:Stroke
language:en
Short-container-title:Stroke

Author:

de Pina-Benabou Mara H.¹,Szostak Vanessa¹,Kyrozis Andreas¹,Rempe David¹,Uziel Daniela¹,Urban-Maldonado Marcia¹,Benabou Salomon¹,Spray David C.¹,Federoff Howard J.¹,Stanton Patric K.¹,Rozental Renato¹

Affiliation:

1. From the Departments of Cell Biology and Anatomy (M.H.dP.-B., P.K.S., R.R.), Neurology (P.K.S.), Obstetrics (R.R.), Pediatrics, and Anesthesiology (R.R.), New York Medical College, Valhalla; Department of Physiology (M.H.dP.-B.), University of São Paulo, Brazil; Department of Neurology (V.S., A.K.) and Neurosciences (M.U.-M., D.C.S.), Albert Einstein College of Medicine, Bronx, NY; Department of Neurology (A.K.), Eginition Hospital, University of Athens, Greece; Department of Anatomy (D.U.),...

Publisher

Ovid Technologies (Wolters Kluwer Health)

Subject

Advanced and Specialized Nursing,Cardiology and Cardiovascular Medicine,Neurology (clinical)

Reference25 articles.

1. Gap Junctions and Neuronal Injury: Protectants or Executioners?

2. Gap-junction-mediated propagation and amplification of cell injury

3. Ischemia-Induced Brain Damage Depends on Specific Gap-Junctional Coupling

4. Specific Gap Junctions Enhance the Neuronal Vulnerability to Brain Traumatic Injury

5. Astrocytic Gap Junctions Remain Open during Ischemic Conditions

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