Deposition of Platelet RANTES Triggering Monocyte Recruitment Requires P-Selectin and Is Involved in Neointima Formation After Arterial Injury-Reference-Cited by-同舟云学术

Deposition of Platelet RANTES Triggering Monocyte Recruitment Requires P-Selectin and Is Involved in Neointima Formation After Arterial Injury

Published:2002-09-17 Issue:12 Volume:106 Page:1523-1529
ISSN:0009-7322
Container-title:Circulation
language:en
Short-container-title:Circulation

Author:

Schober Andreas¹,Manka David¹,von Hundelshausen Philipp¹,Huo Yuqing¹,Hanrath Peter¹,Sarembock Ian J.¹,Ley Klaus¹,Weber Christian¹

Affiliation:

1. From the Departments of Molecular Cardiovascular Research and Cardiology (A.S., P.v.H., P.H., C.W.), Rheinisch-Westfälische Technische Hochschule Aachen, Germany, and Departments of Biomedical Engineering (D.M., Y.H., K.L.) and Medicine (I.J.S.), University of Virginia, Charlottesville. The current address for D.M. is Center for Transgene Technology and Gene Therapy, Leuven, Belgium.

Publisher

Ovid Technologies (Wolters Kluwer Health)

Subject

Physiology (medical),Cardiology and Cardiovascular Medicine

Reference27 articles.

1. Syndromes of accelerated atherosclerosis: Role of vascular injury and smooth muscle cell proliferation

2. Arterial Injury Increases Expression of Inflammatory Adhesion Molecules in the Carotid Arteries of Apolipoprotein-E-Deficient Mice¹

3. Neutrophil accumulation on activated, surface-adherent platelets in flow is mediated by interaction of Mac-1 with fibrinogen bound to alphaIIbbeta3 and stimulated by platelet-activating factor.

4. P-selectin and MAC-1 mediate monocyte rolling and adhesion to ECM-bound platelets under flow conditions

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