BRCA1 Directs a Selective p53-Dependent Transcriptional Response towards Growth Arrest and DNA Repair Targets
Author:
Affiliation:
1. Laboratory of Molecular Oncology and Cell Cycle Regulation
2. Howard Hughes Medical Institute
3. Departments of Medicine
4. Genetics
5. Pharmacology
Publisher
Informa UK Limited
Link
https://journals.asm.org/doi/pdf/10.1128/MCB.22.12.4280-4292.2002
Reference54 articles.
1. BRCA1 Expression Restores Radiation Resistance in BRCA1-defective Cancer Cells through Enhancement of Transcription-coupled DNA Repair
2. Tumor Suppressor p53 Is Required To Modulate BRCA1 Expression
3. PERP, an apoptosis-associated target of p53, is a novel member of the PMP-22/gas3 family
4. Stable Interaction between the Products of the BRCA1 and BRCA2 Tumor Suppressor Genes in Mitotic and Meiotic Cells
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