Expression of Human Endogenous Retrovirus Group K (HERV-K) HML-2 Correlates with Immune Activation of Macrophages and Type I Interferon Response

Author:

Russ Eric123ORCID,Mikhalkevich Natallia12,Iordanskiy Sergey14ORCID

Affiliation:

1. Department of Pharmacology & Molecular Therapeutics, Uniformed Services University of the Health Sciences, Bethesda, Maryland, USA

2. The Henry M. Jackson Foundation for the Advancement of Military Medicine, Bethesda, Maryland, USA

3. Graduate Program of Cellular and Molecular Biology, Uniformed Services University of the Health Sciences, Bethesda, Maryland, USA

4. Armed Forces Radiobiology Research Institute, Uniformed Services University of the Health Sciences, Bethesda, Maryland, USA

Abstract

The human endogenous retrovirus group K subgroup, HML-2, is known to be elevated in a long list of inflammation-associated diseases. However, a clear mechanism for HML-2 upregulation in response to inflammation has not been defined.

Funder

DOD | USA | MEDCOM | Congressionally Directed Medical Research Programs

DOD | USU | Armed Forces Radiobiology Research Institute, Uniformed Services University

DOD | Uniformed Services University of the Health Sciences

The American Genome Research Center

Publisher

American Society for Microbiology

Subject

Infectious Diseases,Cell Biology,Microbiology (medical),Genetics,General Immunology and Microbiology,Ecology,Physiology

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