Cholesterol-Dependent Cytolysins, a Family of Versatile Pore-Forming Toxins
Author:
Affiliation:
1. Department of Microbiology and Immunology, The University of Oklahoma Health Sciences Center, Oklahoma City, Oklahoma 73104
Publisher
American Society for Microbiology
Subject
Infectious Diseases,Immunology,Microbiology,Parasitology
Link
https://journals.asm.org/doi/pdf/10.1128/IAI.73.10.6199-6209.2005
Reference102 articles.
1. Abel, K., M. D. Yoder, R. Hilgenfeld, and F. Jurnak. 1996. An alpha to beta conformational switch in EF-Tu. Structure4:1153-1159.
2. Abrami, L., M. Fivaz, E. Decroly, N. G. Seidah, F. Jean, G. Thomas, S. H. Leppla, J. T. Buckley, and F. G. van der Goot. 1998. The pore-forming toxin proaerolysin is activated by furin. J. Biol. Chem.273:32656-32661.
3. Alving, C. R., W. H. Habig, K. A. Urban, and M. C. Hardegree. 1979. Cholesterol-dependent tetanolysin damage to liposomes. Biochim. Biophys. Acta551:224-228.
4. The Autolytic Enzyme LytA of Streptococcus pneumoniae Is Not Responsible for Releasing Pneumolysin
5. Beauregard, K. E., K. D. Lee, R. J. Collier, and J. A. Swanson. 1997. pH-dependent perforation of macrophage phagosomes by listeriolysin O from Listeria monocytogenes. J. Exp. Med.186:1159-1163.
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