Zika Virus Infection Downregulates Connexin 43, Disrupts the Cardiomyocyte Gap Junctions and Induces Heart Diseases in A129 Mice-Reference-Cited by-全球学者库

Zika Virus Infection Downregulates Connexin 43, Disrupts the Cardiomyocyte Gap Junctions and Induces Heart Diseases in A129 Mice

Published:2022-11-09 Issue:21 Volume:96 Page:
ISSN:0022-538X
Container-title:Journal of Virology
language:en
Short-container-title:J Virol

Author:

Li Shuxuan¹²,Armstrong Najealicka³,Zhao Huan²,Cruz-cosme Ruth³,Yang Hongwei²,Zhong Chunlian⁴,Fu Wenkun²,Wang Wei²,Yang Decheng⁵,Xia Ningshao²,Cheng Tong²,Tang Qiyi³^ORCID

Affiliation:

1. School of Medicine, Henan University of Chinese Medicine, Zhengzhou, P.R. China

2. State Key Laboratory of Molecular Vaccinology and Molecular Diagnostics, National Institute of Diagnostics and Vaccine Development in Infectious Diseases, School of Public Health, School of Life Sciences, Xiamen University, Xiamen, P.R. China

3. Department of Microbiology, Howard University College of Medicine, Washington, DC, USA

4. School of Material and Chemical Engineering, Minjiang University, Fuzhou, P.R. China

5. Centre for Heart Lung Innovation - St. Paul’s Hospital, Department of Pathology and Laboratory Medicine, University of British Columbia, Vancouver, Canada

Abstract

Zika virus (ZIKV) is a teratogen causing devastating sequelae to the newborns who suffer a congenital ZIKV infection while it brings about only mild symptoms to the health-competent older children or adults. Mouse models have played an important role in mechanistic and pathogenic studies of ZIKV.

Publisher

American Society for Microbiology

Subject

Virology,Insect Science,Immunology,Microbiology

Link

https://journals.asm.org/doi/pdf/10.1128/jvi.01373-22

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