Affiliation:
1. Department
of Medicine, Center for Tuberculosis Research, Johns Hopkins School of
Medicine, Baltimore, Maryland 21231
Abstract
ABSTRACT
The
stress-induced extracytoplasmic sigma factor E (SigE) of
Mycobacterium tuberculosis
shows increased expression after
heat shock, sodium dodecyl sulfate treatment, and oxidative stress, as
well as after phagocytosis in macrophages. We report that deletion of
sigE
results in delayed lethality in mice without a
significant reduction of bacterial numbers in
lungs.
Publisher
American Society for Microbiology
Subject
Infectious Diseases,Immunology,Microbiology,Parasitology
Cited by
67 articles.
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