IKKα-Mediated Noncanonical NF-κB Signaling Is Required To Support Murine Gammaherpesvirus 68 LatencyIn Vivo

Author:

Cieniewicz Brandon12,Kirillov Varvara1,Daher Isabel3,Li Xiaofan3,Oldenburg Darby G.4,Dong Qiwen12,Bettke Julie A.2,Marcu Kenneth B.56,Krug Laurie T.13ORCID

Affiliation:

1. Department of Microbiology and Immunology, Stony Brook University, Stony Brook, New York, USA

2. Molecular and Cellular Biology Program, Stony Brook University, Stony Brook, New York, USA

3. HIV and AIDS Malignancy Branch, National Cancer Institute, Bethesda, Maryland, USA

4. Gundersen Medical Foundation, La Crosse, Wisconsin, USA

5. Department of Biochemistry and Cell Biology, Stony Brook University, Stony Brook, New York, USA

6. Department of Pathology, Stony Brook University, Stony Brook, New York, USA

Abstract

The latency programs of the human gammaherpesviruses Epstein-Barr virus (EBV) and Kaposi sarcoma-associated herpesvirus (KSHV) are associated with B cell lymphomas. It is critical to understand the signaling pathways that are used by gammaherpesviruses to establish and maintain latency in primary B cells.

Funder

American Cancer Society

Gundersen Medical Foundation

HHS | NIH | National Cancer Institute

HHS | NIH | National Institute of Allergy and Infectious Diseases

Publisher

American Society for Microbiology

Subject

Virology,Insect Science,Immunology,Microbiology

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