Pathogenesis of Pulmonary Tuberculosis: an Interplay of Tissue-Damaging and Macrophage-Activating Immune Responses-Dual Mechanisms That Control Bacillary Multiplication

Author:

Dannenberg Arthur M.1,Rook Graham A. W.2

Affiliation:

1. Department of Environmental Health Sciences; The Johns Hopkins University School of Hygiene and Public Health; 615 North Wolfe Street, Room 4001 Baltimore Maryland 21205

2. Department of Medical Microbiology; School of Pathology, University College London Medical School; 67-73 Riding House Street London W1P 7LD United Kingdom

Publisher

ASM Press

Reference40 articles.

1. Host-parasite relationships in natively resistant and susceptible rabbits on quantitative inhalation of tubercle bacilli;Allison;Am. Rev. Respir. Dis.,1962

2. Histochemical studies relating the activation of macrophages to the intracellular destruction of tubercle bacilli;Ando;Am. J. Pathol.,1977

3. Growth characteristics of recent sputum isolates of Mycobacterium tuberculosis in guinea pigs infected by the respiratory route;Balasubramanian;Infect. Immun.,1992

4. Human Mycobacterium tuberculosis-reactive CD4+ T-cell clones: heterogeneity in antigen recognition, cytokine production, and cytotoxicity for mononuclear phagocytes;Boom;Infect. Immun.,1991

5. Canetti , G. 1955 The Tubercle Bacillus in the Pulmonary Lesion of Man 130 Springer Publishing Co., Inc New York

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