Interaction of Helicobacter pylori and its fatty acids with parietal cells and gastric H+/K(+)-ATPase.

Author:

Beil W,Birkholz C,Wagner S,Sewing K F

Publisher

BMJ

Subject

Gastroenterology

Reference16 articles.

1. Campylobacter pyloridis causes gastritis and raised fasting gastric pH;Morris, A.; Nicholson, G.;Am J Gastroenterol,1987

2. Iatrogenic Campylobacter pylori infection is a cause of epidemic achlorhydria;Graham, D.Y.; Alpert, L.C.; Smith, J.L.; Yoshimura, H.H.;Am J Gastroenterol,1988

3. Interleukin1 is cytoprotective, antisecretory, stimulates PGE2 synthesis by the stomach and retards gastric emptying;Robert, A.; Olafsson, A.S.; Lancaster, C.; Zhang, W.;Life Sci,1991

4. Effect of a Campylobacter pylori protein on acid secretion by parietal cells;Cave, D.R.; Vargas, M.;Lancet,1989

5. Unusual cellular fatty acids and distinctive ultrastructure in a new spiral bacterium (Campylobacter pyloridis) from the human gastric mucosa;Goodwin, C.S.; McCulloch, R.K.; Armstrong, J.A.; Wee, S.H.;J Med Microbiol,1985

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