Okamoto model for necrosis and its expansions, CD38-cyclic ADP-ribose signal system for intracellular Ca2+ mobilization and Reg (Regenerating gene protein)-Reg receptor system for cell regeneration

Author:

OKAMOTO Hiroshi1,TAKASAWA Shin2

Affiliation:

1. Department of Biochemistry, Tohoku University Graduate School of Medicine

2. Department of Biochemistry, Nara Medical University

Publisher

Japan Academy

Subject

General Physics and Astronomy,General Agricultural and Biological Sciences,General Medicine

Reference295 articles.

1. 1) Okamoto, H. (ed.) (1990 & 2008) Molecular Biology of the Islets of Langerhans. Cambridge University Press, Cambridge, New York, Port Chester, Melbourne, Sydney.

2. 2) Okamoto, H. (1981) Regulation of proinsulin synthesis in pancreatic islets and a new aspect to insulin-dependent diabetes. Mol. Cell. Biochem. 37, 43–61.

3. 3) Steiner, D.F., Kemmler, W., Clark, J.L., Oyer, P.E. and Rubenstein, A.H. (1972) The biosynthesis of insulin. In Handbook of Physiology, section 7, vol. 1 (eds. Steiner, D.F. and Freinkel, N.). American Physiology Society, Washington, D.C., pp. 175–198.

4. 4) Yamamoto, H., Uchigata, Y. and Okamoto, H. (1981) Streptozotocin and alloxan induce DNA strand breaks and poly(ADP-ribose) synthetase in pancreatic islets. Nature 294, 284–286.

5. 5) Yamamoto, H., Uchigata, Y. and Okamoto, H. (1981) DNA strand breaks in pancreatic islets by in vivo administration of alloxan or streptozotocin. Biochem. Biophys. Res. Commun. 103, 1014–1020.

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