Does the prenatal bisphenol A exposure alter DNA methylation levels in the mouse hippocampus?: An analysis using a high-sensitivity methylome technique
Author:
Funder
Japan Society for the Promotion of Science
Publisher
Springer Science and Business Media LLC
Subject
Environmental Science (miscellaneous),Genetics,Social Psychology
Link
http://link.springer.com/content/pdf/10.1186/s41021-018-0099-y.pdf
Reference44 articles.
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2. van der Meer TP, Artacho-Cordón F, Swaab DF, Struik D, Makris KC, Wolffenbuttel BHR, et al. Distribution of non-persistent endocrine disruptors in two different regions of the human brain. Int. J. Environ. Res. Public Health [Internet]. 2017;14:1059. Available from: http://www.mdpi.com/1660-4601/14/9/1059
3. Arase S, Ishii K, Igarashi K, Aisaki K, Yoshio Y, Matsushima A, et al. Endocrine disrupter bisphenol a increases in situ estrogen production in the mouse urogenital sinus. Biol Reprod [Internet]. 2011;84:734–42. Available from: http://www.ncbi.nlm.nih.gov/pubmed/21123812
4. Liu X, Matsushima A, Shimohigashi M, Shimohigashi Y. A characteristic back support structure in the bisphenol A-binding pocket in the human nuclear receptor ERRγ. Vanacker J-M, editor. PLoS One [Internet]. 2014;9:e101252. Available from: http://www.ncbi.nlm.nih.gov/pubmed/24978476
5. Sartain CV, Hunt PA. An old culprit but a new story: bisphenol a and “NextGen” bisphenols. Fertil Steril [Internet]. 2016;106:820–6. Available from: http://www.ncbi.nlm.nih.gov/pubmed/27504789
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