Complement C3a/C3aR inhibition alleviates the formation of aortic aneurysm in Marfan syndrome mice
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Publisher
Springer Science and Business Media LLC
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https://link.springer.com/content/pdf/10.1186/s12872-024-04077-6.pdf
Reference43 articles.
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2. Collod-Béroud G, Le Bourdelles S, Ades L, Ala-Kokko L, Booms P, Boxer M, Child A, Comeglio P, De Paepe A, Hyland J, et al. Update of the UMD-FBN1 mutation database and creation of an FBN1 polymorphism database. Hum Mutat. 2003;22(3):199–208.
3. Judge D, Biery N, Keene D, Geubtner J, Myers L, Huso D, Sakai L, Dietz H. Evidence for a critical contribution of haploinsufficiency in the complex pathogenesis of Marfan syndrome. J Clin Investig. 2004;114(2):172–81.
4. Cook J, Clayton N, Carta L, Galatioto J, Chiu E, Smaldone S, Nelson C, Cheng S, Wentworth B, Ramirez F. Dimorphic effects of transforming growth factor-β signaling during aortic aneurysm progression in mice suggest a combinatorial therapy for Marfan syndrome. Arterioscler Thromb Vasc Biol. 2015;35(4):911–7.
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