Presence of hepatitis B virus in synovium and its clinical significance in rheumatoid arthritis-Reference-Cited by-全球学者库

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Presence of hepatitis B virus in synovium and its clinical significance in rheumatoid arthritis

Published:2018-06-19 Issue:1 Volume:20 Page:
ISSN:1478-6362
Container-title:Arthritis Research & Therapy
language:en
Short-container-title:Arthritis Res Ther

Author:

Chen Yu-Lan,Jing Jun,Mo Ying-Qian,Ma Jian-Da,Yang Li-Juan,Chen Le-Feng,Zhang Xiang,Yan Tao,Zheng Dong-Hui,Pessler Frank,Dai Lie

Funder

National Natural Science Foundation of China

Natural Science Foundation of Guangdong Province

Fundamental Research Funds for the Central Universities

Helmholtz Association

Publisher

Springer Science and Business Media LLC

Link

http://link.springer.com/content/pdf/10.1186/s13075-018-1623-y.pdf

Reference47 articles.

1. McInnes IB, Schett G. Pathogenetic insights from the treatment of rheumatoid arthritis. Lancet. 2017;389:2328–37.

2. Ebringer A, Wilson C. HLA molecules, bacteria and autoimmunity. J Med Microbiol. 2000;49:305–11.

3. Alam J, Jantan I, Bukhari SNA. Rheumatoid arthritis: recent advances on its etiology, role of cytokines and pharmacotherapy. Biomed Pharmacother. 2017;92:615–33.

4. Neuveut C, Yu W, Buendia MA. Mechanisms of HBV-related hepatocarcinogenesis. J Hepatol. 2010;52:594–604.

5. Mason A, Theal J, Bain V, Adams E, Perrillo R. Hepatitis B virus replication in damaged endothelial tissues of patients with extrahepatic disease. Am J Gastroenterol. 2005;100:972–6.

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4. How long is long COVID;International Journal of Rheumatic Diseases;2022-12-05

5. Mucin 1 aggravates synovitis and joint damage of rheumatoid arthritis by regulating inflammation and aggression of fibroblast-like synoviocytes;Bone & Joint Research;2022-09-01

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