Mechanisms of ARIA: is it time to focus on the unique immune environment of the neurovascular unit?
Author:
Funder
NINDS
BrightFocus Foundation
Publisher
Springer Science and Business Media LLC
Subject
Cellular and Molecular Neuroscience,Neurology (clinical),Molecular Biology
Link
https://link.springer.com/content/pdf/10.1186/s13024-023-00667-8.pdf
Reference11 articles.
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2. Wilcock DM, Munireddy SK, Rosenthal A, Ugen KE, Gordon MN, Morgan D. Microglial activation facilitates abeta plaque removal following intracranial anti-abeta antibody administration. Neurobiol Dis. 2004;15(1):11–20.
3. Racke MM, Boone LI, Hepburn DL, Parsadainian M, Bryan MT, Ness DK, et al. Exacerbation of cerebral amyloid angiopathy-associated microhemorrhage in amyloid precursor protein transgenic mice by immunotherapy is dependent on antibody recognition of deposited forms of amyloid beta. J Neurosci. 2005;25(3):629–36.
4. Sperling RA, Jack CR Jr., Black SE, Frosch MP, Greenberg SM, Hyman BT, et al. Amyloid-related imaging abnormalities in amyloid-modifying therapeutic trials: recommendations from the Alzheimer’s Association Research Roundtable Workgroup. Alzheimers Dement. 2011;7(4):367–85.
5. Taylor X, Clark IM, Fitzgerald GJ, Oluoch H, Hole JT, DeMattos RB, et al. Amyloid-β (Aβ) immunotherapy induced microhemorrhages are associated with activated perivascular macrophages and peripheral monocyte recruitment in Alzheimer’s Disease mice. Mol Neurodegeneration. 2023;18(1):59.
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1. Risk factors in developing amyloid related imaging abnormalities (ARIA) and clinical implications;Frontiers in Neuroscience;2024-01-19
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