Contribution of 1425G/A Polymorphism in Protein Kinase C-Eta (PRKCH) Gene and Brain White Matter Lesions to the Risk of Sudden Sensorineural Hearing Loss in a Japanese Nested Case-Control Study-Reference-Cited by-同舟云学术

Contribution of 1425G/A Polymorphism in Protein Kinase C-Eta (PRKCH) Gene and Brain White Matter Lesions to the Risk of Sudden Sensorineural Hearing Loss in a Japanese Nested Case-Control Study

Published:2011-07-14 Issue:3 Volume:25 Page:82-87
ISSN:0167-7063
Container-title:Journal of Neurogenetics
language:en
Short-container-title:Journal of Neurogenetics

Author:

Uchida Yasue¹²,Sugiura Saiko¹,Nakashima Tsutomu³,Ando Fujiko⁴⁵,Shimokata Hiroshi⁵

Affiliation:

1. Department of Otorhinolaryngology, National Center for Geriatrics and Gerontology, Obu City, Aichi Prefecture, Japan

2. Department of Otorhinolaryngology, Aichi Medical University, Nagakute, Aichi Prefecture, Japan

3. Department of Otorhinolaryngology Cognitive and Speech Medicine, Nagoya University School of Medicine, Nagoya City, Aichi Prefecture, Japan

4. Department of Health and Medical Sciences, Aichi Shukutoku University, Aichi Prefecture, Japan

5. Department for Development of Preventive Medicine, National Center for Geriatrics and Gerontology, Obu City, Aichi Prefecture, Japan

Publisher

Informa UK Limited

Subject

Cellular and Molecular Neuroscience,Genetics

Link

https://www.tandfonline.com/doi/pdf/10.3109/01677063.2011.591462

Reference18 articles.

1. Magnetic Resonance Imaging Findings in Sudden Sensorineural Hearing Loss

2. Pathologic correlates of incidental MRI white matter signal hyperintensities

3. Sudden deafness without vertigo as a sole manifestation of AICA infarct

4. MTHFR Gene Polymorphism as a Risk Factor for Silent Brain Infarcts and White Matter Lesions in the Japanese General Population

5. A nonsynonymous SNP in PRKCH (protein kinase C η) increases the risk of cerebral infarction

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