Boldly Targeting Kinases without mutations
Author:
Affiliation:
1. DANA-FARBER CANCER INSTITUTE
Publisher
American Society of Hematology
Subject
Cell Biology,Hematology,Immunology,Biochemistry
Link
http://ashpublications.org/blood/article-pdf/123/8/1119/1378812/1119.pdf
Reference13 articles.
1. Bruton’s tyrosine kinase (BTK) function is important to the development and expansion of chronic lymphocytic leukemia (CLL).;Woyach;Blood,2014
2. Identification of Bruton’s tyrosine kinase as a therapeutic target in acute myeloid leukemia.;Rushworth;Blood,2014
3. Five-year follow-up of patients receiving imatinib for chronic myeloid leukemia.;Druker;N Engl J Med,2006
4. Improved survival with vemurafenib in melanoma with BRAF V600E mutation.;Chapman;N Engl J Med,2011
5. Anaplastic lymphoma kinase inhibition in non-small-cell lung cancer.;Kwak;N Engl J Med,2010
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2. Bruton’s tyrosine kinase inhibition increases BCL-2 dependence and enhances sensitivity to venetoclax in chronic lymphocytic leukemia;Leukemia;2017-01-23
3. The Development and Current Use of BCL-2 Inhibitors for the Treatment of Chronic Lymphocytic Leukemia;Current Hematologic Malignancy Reports;2017-01-23
4. Pharmacokinetic and pharmacodynamic evaluation of ibrutinib for the treatment of chronic lymphocytic leukemia: rationale for lower doses;Expert Opinion on Drug Metabolism & Toxicology;2016-10-11
5. Emerging therapeutic targets for the treatment of human acute myeloid leukemia (part 1) – gene transcription, cell cycle regulation, metabolism and intercellular communication;Expert Review of Hematology;2015-04-03
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