Functional Differences Between Direct and Indirect Striatal Output Pathways in Huntington's Disease
Author:
Affiliation:
1. Intellectual and Developmental Disabilities Research Center, Semel Institute for Neuroscience and Human Behavior and the Brain Research Institute, David Geffen School of Medicine, University of California Los Angeles, Los Angeles, CA, USA
Publisher
IOS Press
Subject
Cellular and Molecular Neuroscience,Clinical Neurology
Cited by 49 articles. 订阅此论文施引文献 订阅此论文施引文献,注册后可以免费订阅5篇论文的施引文献,订阅后可以查看论文全部施引文献
1. Pharmacological Co-Activation of TrkB and TrkC Receptor Signaling Ameliorates Striatal Neuropathology and Motor Deficits in Mouse Models of Huntington’s Disease;Journal of Huntington's Disease;2023-10-03
2. The many “Neurofaces” of Prohibitins 1 and 2: Crucial for the healthy brain, dysregulated in numerous brain disorders;Journal of Chemical Neuroanatomy;2023-10
3. A bird's eye view on cellular dynamics in Huntington's disease;CURR SCI INDIA;2023
4. Case report: Cerebellar sparing in juvenile Huntington's disease;Frontiers in Neurology;2023-01-11
5. Hunting for the cause: Evidence for prion-like mechanisms in Huntington’s disease;Frontiers in Neuroscience;2022-08-24
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