Liquid Chromatography–Tandem Mass Spectrometry Method for the Analysis of Asymmetric Dimethylarginine in Human Plasma
Author:
Affiliation:
1. Institute of Experimental and Clinical Pharmacology, University Hospital Hamburg-Eppendorf, and
2. Institute of Pharmacy, University of Hamburg, Hamburg, Germany
Publisher
Oxford University Press (OUP)
Subject
Biochemistry, medical,Clinical Biochemistry
Link
http://academic.oup.com/clinchem/article-pdf/51/7/1268/32683805/clinchem1268.pdf
Reference17 articles.
1. Tsikas D, Böger RH, Sandmann J, Bode-Böger SM, Frölich JC. Endogenous nitric oxide synthase inhibitors are responsible for the l-arginine paradox. FEBS Lett2000;478:1-3.
2. McBride AE, Silver PA. State of the Arg: protein methylation at arginine comes of age. Cell2001;106:5-8.
3. Achan V, Broadhead M, Malaki M, Whitley G, Leiper J, MacAllister R, et al. Asymmetric dimethylarginine causes hypertension and cardiac dysfunction in humans and is actively metabolized by dimethylarginine dimethylaminohydrolase. Arterioscler Thromb Vasc Biol2003;23:1455-1459.
4. Böger RH, Bode-Böger SM, Szuba A, Tsao PS, Chan JR, Tangphao O, et al. Asymmetric dimethylarginine (ADMA): a novel risk factor for endothelial dysfunction: its role in hypercholesterolemia. Circulation1998;98:1842-1847.
5. Selley ML. Increased concentrations of homocysteine and asymmetric dimethylarginine and decreased concentrations of nitric oxide in the plasma of patients with Alzheimer’s disease. Neurobiol Aging2003;24:903-907.
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