The promotion of fatty acid β-oxidation by hesperidin <i>via</i> activating SIRT1/PGC1α to improve NAFLD induced by a high-fat diet-Reference-Cited by-全球学者库

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The promotion of fatty acid β-oxidation by hesperidin via activating SIRT1/PGC1α to improve NAFLD induced by a high-fat diet

Published:2024 Issue:1 Volume:15 Page:372-386
ISSN:2042-6496
Container-title:Food & Function
language:en
Short-container-title:Food Funct.

Author:

Nie Tong¹^ORCID,Wang Xin¹,Li Aqun¹,Shan Anshan²,Ma Jun¹³

Affiliation:

1. College of Veterinary Medicine, Northeast Agricultural University, Harbin, 150030, P. R. China

2. College of Animal Science and Technology, Northeast Agricultural University, Harbin, 150030, P. R. China

3. Heilongjiang Provincial Key Laboratory of Pathogenic Mechanism for Animal Disease and Comparative Medicine, Harbin, 150030, P.R. China

Abstract

Reducing fat deposits in hepatocytes is a direct treatment for nonalcoholic fatty liver disease (NAFLD) and the fatty acid metabolic processes mediated by fatty acid β-oxidation are important for the prevention of NAFLD.

Publisher

Royal Society of Chemistry (RSC)

Subject

General Medicine,Food Science

Link

http://pubs.rsc.org/en/content/articlepdf/2024/FO/D3FO04348G

Reference69 articles.

1. Non-alcoholic fatty liver disease (NAFLD) – pathogenesis, classification, and effect on drug metabolizing enzymes and transporters

2. Microbiota, Obesity and NAFLD

3. Animal models of NAFLD from a hepatologist's point of view

4. NAFLD and increased risk of cardiovascular disease: clinical associations, pathophysiological mechanisms and pharmacological implications

5. Current treatment paradigms and emerging therapies for NAFLD/NASH

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