Involvement of histone hypoacetylation in INH-induced rat liver injury
Author:
Affiliation:
1. Hebei Province Key Laboratory of Occupational Health and Safety for Coal Industry
2. School of Public Health
3. North China University of Science and Technology
4. Tangshan 063210
5. China
Abstract
This study explores the mechanism of histone acetylation under the effect of oxidative stress in rat liver injury induced by isoniazid (INH).
Publisher
Oxford University Press (OUP)
Subject
Health, Toxicology and Mutagenesis,Toxicology
Link
http://pubs.rsc.org/en/content/articlepdf/2018/TX/C7TX00166E
Reference22 articles.
1. Therapeutic potential of thymoquinone against anti-tuberculosis drugs induced liver damage
2. Mechanisms of isoniazid-induced idiosyncratic liver injury: Emerging role of mitochondrial stress
3. Hepatocyte Growth Factor Protects Against Isoniazid/Rifampicin-Induced Oxidative Liver Damage
4. Role of hydrazine in isoniazid-induced hepatotoxicity in a hepatocyte inflammation model
5. Oxidative Stress and Chromatin Remodeling in Chronic Obstructive Pulmonary Disease and Smoking-Related Diseases
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