Frequent pathway mutations of splicing machinery in myelodysplasia-Reference-Cited by-同舟云学术

Frequent pathway mutations of splicing machinery in myelodysplasia

Published:2011-09-11 Issue:7367 Volume:478 Page:64-69
ISSN:0028-0836
Container-title:Nature
language:en
Short-container-title:Nature

Author:

Yoshida Kenichi,Sanada Masashi,Shiraishi Yuichi,Nowak Daniel,Nagata Yasunobu,Yamamoto Ryo,Sato Yusuke,Sato-Otsubo Aiko,Kon Ayana,Nagasaki Masao,Chalkidis George,Suzuki Yutaka,Shiosaka Masashi,Kawahata Ryoichiro,Yamaguchi Tomoyuki,Otsu Makoto,Obara Naoshi,Sakata-Yanagimoto Mamiko,Ishiyama Ken,Mori Hiraku,Nolte Florian,Hofmann Wolf-Karsten,Miyawaki Shuichi,Sugano Sumio,Haferlach Claudia,Koeffler H. Phillip,Shih Lee-Yung,Haferlach Torsten,Chiba Shigeru,Nakauchi Hiromitsu,Miyano Satoru,Ogawa Seishi

Publisher

Springer Science and Business Media LLC

Subject

Multidisciplinary

Link

http://www.nature.com/articles/nature10496.pdf

Reference37 articles.

1. Corey, S. J. et al. Myelodysplastic syndromes: the complexity of stem-cell diseases. Nature Rev. Cancer 7, 118–129 (2007)

2. Ma, X., Does, M., Raza, A. & Mayne, S. T. Myelodysplastic syndromes: incidence and survival in the United States. Cancer 109, 1536–1542 (2007)

3. Bejar, R., Levine, R. & Ebert, B. L. Unraveling the molecular pathophysiology of myelodysplastic syndromes. J. Clin. Oncol. 29, 504–515 (2011)

4. Sanada, M. et al. Gain-of-function of mutated C-CBL tumour suppressor in myeloid neoplasms. Nature 460, 904–908 (2009)

5. Campbell, P. J. et al. Identification of somatically acquired rearrangements in cancer using genome-wide massively parallel paired-end sequencing. Nature Genet. 40, 722–729 (2008)

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