p75NTR ectodomain is a physiological neuroprotective molecule against amyloid-beta toxicity in the brain of Alzheimer’s disease-Reference-Cited by-全球学者库

p75NTR ectodomain is a physiological neuroprotective molecule against amyloid-beta toxicity in the brain of Alzheimer’s disease

Published:2015-04-28 Issue:11 Volume:20 Page:1301-1310
ISSN:1359-4184
Container-title:Molecular Psychiatry
language:en
Short-container-title:Mol Psychiatry

Author:

Yao X-Q,Jiao S-S,Saadipour K,Zeng F,Wang Q-H,Zhu C,Shen L-L,Zeng G-H,Liang C-R,Wang J^ORCID,Liu Y-H,Hou H-Y,Xu X,Su Y-P,Fan X-T,Xiao H-L,Lue L-F,Zeng Y-Q,Giunta B,Zhong J-H,Walker D G,Zhou H-D,Tan J,Zhou X-F,Wang Y-J

Publisher

Springer Science and Business Media LLC

Subject

Cellular and Molecular Neuroscience,Psychiatry and Mental health,Molecular Biology

Link

http://www.nature.com/articles/mp201549.pdf

Reference49 articles.

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3. Castellani RJ, Perry G . Pathogenesis and disease-modifying therapy in Alzheimer's disease: the flat line of progress. Arch Med Res 2012; 43: 694–698.

4. Garzon DJ, Fahnestock M . Oligomeric amyloid decreases basal levels of brain-derived neurotrophic factor (BDNF) mRNA via specific downregulation of BDNF transcripts IV and V in differentiated human neuroblastoma cells. J Neurosci 2007; 27: 2628–2635.

5. Pedraza CE, Podlesniy P, Vidal N, Arevalo JC, Lee R, Hempstead B et al. Pro-NGF isolated from the human brain affected by Alzheimer's disease induces neuronal apoptosis mediated by p75NTR. Am J Pathol 2005; 166: 533–543.

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