BMI1 represses Ink4a/Arf and Hox genes to regulate stem cells in the rodent incisor-Reference-Cited by-同舟云学术

BMI1 represses Ink4a/Arf and Hox genes to regulate stem cells in the rodent incisor

Published:2013-06-02 Issue:7 Volume:15 Page:846-852
ISSN:1465-7392
Container-title:Nature Cell Biology
language:en
Short-container-title:Nat Cell Biol

Author:

Biehs Brian,Hu Jimmy Kuang-Hsien,Strauli Nicolas B.,Sangiorgi Eugenio,Jung Heekyung,Heber Ralf-Peter,Ho Sunita,Goodwin Alice F.,Dasen Jeremy S.,Capecchi Mario R.,Klein Ophir D.

Publisher

Springer Science and Business Media LLC

Subject

Cell Biology

Link

http://www.nature.com/articles/ncb2766.pdf

Reference39 articles.

1. Molofsky, A. V. et al. Bmi-1 dependence distinguishes neural stem cell self-renewal from progenitor proliferation. Nature 425, 962–967 (2003).

2. Park, I. K. et al. Bmi-1 is required for maintenance of adult self-renewing haematopoietic stem cells. Nature 423, 302–305 (2003).

3. Jacobs, J. J., Kieboom, K., Marino, S., DePinho, R. A. & van Lohuizen, M. The oncogene and Polycomb-group gene bmi-1 regulates cell proliferation and senescence through the ink4a locus. Nature 397, 164–168 (1999).

4. Molofsky, A. V., He, S., Bydon, M., Morrison, S. J. & Pardal, R. Bmi-1 promotes neural stem cell self-renewal and neural development but not mouse growth and survival by repressing the p16Ink4a and p19Arf senescence pathways. Genes Dev. 19, 1432–1437 (2005).

5. Zacharek, S. J. et al. Lung stem cell self-renewal relies on BMI1-dependent control of expression at imprinted loci. Cell Stem Cell 9, 272–281 (2011).

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