Akt promotes chemoresistance in human ovarian cancer cells by modulating cisplatin-induced, p53-dependent ubiquitination of FLICE-like inhibitory protein-Reference-Cited by-同舟云学术

Akt promotes chemoresistance in human ovarian cancer cells by modulating cisplatin-induced, p53-dependent ubiquitination of FLICE-like inhibitory protein

Published:2009-10-05 Issue:1 Volume:29 Page:11-25
ISSN:0950-9232
Container-title:Oncogene
language:en
Short-container-title:Oncogene

Author:

Abedini M R,Muller E J,Bergeron R,Gray D A,Tsang B K

Publisher

Springer Science and Business Media LLC

Subject

Cancer Research,Genetics,Molecular Biology

Link

http://www.nature.com/articles/onc2009300.pdf

Reference38 articles.

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2. Abedini MR, Qiu Q, Yan X, Tsang BK . (2004). Possible role of FLICE-like inhibitory protein (FLIP) in chemoresistant ovarian cancer cells in vitro. Oncogene 23: 6997–7004.

3. Alladina SJ, Song JH, Davidge ST, Hao C, Easton AS . (2005). TRAIL-induced apoptosis in human vascular endothelium is regulated by phosphatidylinositol 3-kinase/Akt through the short form of cellular FLIP and Bcl-2. J Vasc Res 42: 337–347.

4. Buttitta F, Marchetti A, Gadducci A, Pellegrini S, Morganti M, Carnicelli V et al. (1997). p53 alterations are predictive of chemoresistance and aggressiveness in ovarian carcinomas: a molecular and immunohistochemical study. Br J Cancer 75: 230–235.

5. Chandrasekaran Y, McKee CM, Ye Y, Richburg JH . (2006). Influence of TRP53 status on FAS membrane localization, CFLAR (c-FLIP) ubiquitinylation, and sensitivity of GC-2spd (ts) cells to undergo FAS-mediated apoptosis. Biol Reprod 74: 560–568.

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