Iron, brain ageing and neurodegenerative disorders
Author:
Publisher
Springer Science and Business Media LLC
Subject
General Neuroscience
Link
http://www.nature.com/articles/nrn1537.pdf
Reference137 articles.
1. Crichton, R. R. Inorganic Biochemistry of Iron Metabolism: From Molecular Mechanisms to Clinical Consequences (John Wiley & Sons, Chichester, 2001) A comprehensive overview of iron biochemistry, from molecules to medicine.
2. Pigeon, C. et al. A new mouse liver-specific gene, encoding a protein homologous to human antimicrobial peptide hepcidin, is overexpressed during iron overload. J. Biol. Chem. 276, 7811–7819 (2001).
3. Nicolas, G. et al. Lack of hepcidin gene expression and severe tissue iron overload in upstream stimulatory factor 2 (USF2) knockout mice. Proc. Natl Acad. Sci. USA 98, 8780–8785 (2001).
4. Papanikolaou, G. et al. Mutations in HFE2 cause iron overload in chromosome 1q-linked juvenile hemochromatosis. Nature Genet. 36, 77–82 (2004).
5. Andrews, N. C. Iron homeostasis: insights from genetics and animal models. Nature Rev. Genet. 1, 208–217 (2000).
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