Clostridium difficile toxins induce VEGF-A and vascular permeability to promote disease pathogenesis
Author:
Publisher
Springer Science and Business Media LLC
Subject
Cell Biology,Microbiology (medical),Genetics,Applied Microbiology and Biotechnology,Immunology,Microbiology
Link
http://www.nature.com/articles/s41564-018-0300-x.pdf
Reference52 articles.
1. Bartlett, J. G. Clostridium difficile: history of its role as an enteric pathogen and the current state of knowledge about the organism. Clin. Infect. Dis. 18, S265–S272 (1994).
2. Reineke, J. et al. Autocatalytic cleavage of Clostridium difficile toxin B. Nature 446, 415–419 (2007).
3. Torres, J., Jennische, E., Lange, S. & Lonnroth, I. Enterotoxins from Clostridium difficile; diarrhoeogenic potency and morphological effects in the rat intestine. Gut 31, 781–785 (1990).
4. Mitchell, T. J. et al. Effect of toxin A and B of Clostridium difficile on rabbit ileum and colon. Gut 27, 78–85 (1986).
5. Lyras, D. et al. Toxin B is essential for virulence of Clostridium difficile. Nature 458, 1176–1179 (2009).
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