GATA4 mutations cause human congenital heart defects and reveal an interaction with TBX5-Reference-Cited by-同舟云学术

GATA4 mutations cause human congenital heart defects and reveal an interaction with TBX5

Published:2003-07 Issue:6947 Volume:424 Page:443-447
ISSN:0028-0836
Container-title:Nature
language:en
Short-container-title:Nature

Author:

Garg Vidu,Kathiriya Irfan S.,Barnes Robert,Schluterman Marie K.,King Isabelle N.,Butler Cheryl A.,Rothrock Caryn R.,Eapen Reenu S.,Hirayama-Yamada Kayoko,Joo Kunitaka,Matsuoka Rumiko,Cohen Jonathan C.,Srivastava Deepak

Publisher

Springer Science and Business Media LLC

Subject

Multidisciplinary

Link

http://www.nature.com/articles/nature01827.pdf

Reference30 articles.

1. Hoffman, J. I. E. Incidence of congenital heart disease: I. Postnatal incidence. Pediatr. Cardiol. 16, 103–113 (1995)

2. Schott, J. J. et al. Congenital heart disease caused by mutations in the transcription factor NKX2–5. Science 281, 108–111 (1998)

3. Srivastava, D. & Olson, E. N. A genetic blueprint for cardiac development. Nature 407, 221–226 (2000)

4. Molkentin, J. D., Lin, Q., Duncan, S. A. & Olson, E. N. Requirement of the transcription factor GATA4 for heart tube formation and ventral morphogenesis. Genes Dev. 11, 1061–1072 (1997)

5. Kuo, C. T. et al. GATA4 transcription factor is required for ventral morphogenesis and heart tube formation. Genes Dev. 11, 1048–1060 (1997)

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