Telomeres in cancer: tumour suppression and genome instability
Author:
Publisher
Springer Science and Business Media LLC
Subject
Cell Biology,Molecular Biology
Link
http://www.nature.com/articles/nrm.2016.171.pdf
Reference185 articles.
1. Artandi, S. E. et al. Telomere dysfunction promotes non-reciprocal translocations and epithelial cancers in mice. Nature 406, 641–645 (2000). Demonstrates that telomere attrition in p53-mutant mice promotes epithelial cancers through the formation of chromosome rearrangements.
2. Artandi, S. E. & DePinho, R. A. Telomeres and telomerase in cancer. Carcinogenesis 31, 9–18 (2010).
3. Maciejowski, J., Li, Y., Bosco, N., Campbell, P. J. & de Lange, T. Chromothripsis and kataegis induced by telomere crisis. Cell 163, 1641–1654 (2015). Shows that dicentric chromosomes formed during telomere crisis persist through mitosis, are fragmented by TREX1 in G1 phase and give rise to chromothripsis and kataegis.
4. Hanahan, D. & Weinberg, R. A. Hallmarks of cancer: the next generation. Cell 144, 646–674 (2011).
5. Yates, L. R. & Campbell, P. J. Evolution of the cancer genome. Nat. Rev. Genet. 13, 795–806 (2012).
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