Overexpression of an isoform of AML1 in acute leukemia and its potential role in leukemogenesis
Author:
Publisher
Springer Science and Business Media LLC
Subject
Oncology,Cancer Research,Hematology
Link
http://www.nature.com/articles/leu2008350.pdf
Reference31 articles.
1. Miyoshi H, Ohira M, Shimizu K, Mitani K, Hirai H, Imai T et al. Alternative splicing and genomic structure of the AML1 gene involved in acute myeloid leukemia. Nucleic Acids Res 1995; 23: 2762–2769.
2. Miyoshi H, Shimizu K, Kozu T, Maseki N, Kaneko Y, Ohki M . t(8;21) breakpoints on chromosome 21 in acute myeloid leukemia are clustered within a limited region of a single gene, AMLl. Proc Natl Acad Sci USA 1991; 88: 10431–10434.
3. Nucifora G, Birn DJ, Espinosa R, Erickson P, LeBeau MM, Roulston D et al. Involvement of the AML1 gene in therapy-related leukemia and in chronic myeloid leukemia in blast crisis. Blood 1993; 81: 2728–2734.
4. Golub TR, Barker GF, Bohlander SK, Hiebert SW, Ward DC, Bray-Ward P et al. Fusion of the Tel gene on 12p13 to the AML1 on 21q22 in acute lymphoblastic leukemia. Proc Natl Acad Sci USA 1995; 92: 4917–4921.
5. Meyers S, Lenny N, Hiebert SW . The t (8; 21) fusion protein interferes with AML-1B-dependent transcnptional activation. Mol cell Biol 1995; 15: 1974–1982.
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