ATP13A2 deficiency disrupts lysosomal polyamine export-Reference-Cited by-同舟云学术

ATP13A2 deficiency disrupts lysosomal polyamine export

Published:2020-01-29 Issue:7795 Volume:578 Page:419-424
ISSN:0028-0836
Container-title:Nature
language:en
Short-container-title:Nature

Author:

van Veen Sarah,Martin Shaun,Van den Haute Chris,Benoy Veronick,Lyons Joseph,Vanhoutte Roeland,Kahler Jan Pascal,Decuypere Jean-Paul,Gelders Géraldine,Lambie Eric,Zielich Jeffrey,Swinnen Johannes V.,Annaert Wim,Agostinis Patrizia,Ghesquière Bart,Verhelst Steven,Baekelandt Veerle,Eggermont Jan,Vangheluwe Peter

Publisher

Springer Science and Business Media LLC

Subject

Multidisciplinary

Link

http://www.nature.com/articles/s41586-020-1968-7.pdf

Reference60 articles.

1. Ramirez, A. et al. Hereditary parkinsonism with dementia is caused by mutations in ATP13A2, encoding a lysosomal type 5 P-type ATPase. Nat. Genet. 38, 1184–1191 (2006).

2. Di Fonzo, A. et al. ATP13A2 missense mutations in juvenile parkinsonism and young onset Parkinson disease. Neurology 68, 1557–1562 (2007).

3. Dehay, B. et al. Loss of P-type ATPase ATP13A2/PARK9 function induces general lysosomal deficiency and leads to Parkinson disease neurodegeneration. Proc. Natl Acad. Sci. USA 109, 9611–9616 (2012).

4. Palmgren, M. G. & Nissen, P. P-type ATPases. Annu. Rev. Biophys. 40, 243–266 (2011).

5. van Veen, S. et al. Cellular function and pathological role of ATP13A2 and related P-type transport ATPases in Parkinson’s disease and other neurological disorders. Front. Mol. Neurosci. 7, 48 (2014).

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