Behavioral and synaptic alterations relevant to obsessive-compulsive disorder in mice with increased EAAT3 expression

Author:

Delgado-Acevedo Claudia,Estay Sebastián F.ORCID,Radke Anna K.,Sengupta AyeshaORCID,Escobar Angélica P.,Henríquez-Belmar Francisca,Reyes Cristopher A.,Haro-Acuña Valentina,Utreras Elías,Sotomayor-Zárate Ramón,Cho Andrew,Wendland Jens R.ORCID,Kulkarni Ashok B.,Holmes Andrew,Murphy Dennis L.,Chávez Andrés E.ORCID,Moya Pablo R.

Funder

Ministerio de Economía, Fomento y Turismo

U.S. Department of Health & Human Services | NIH | National Institute of Dental and Craniofacial Research

U.S. Department of Health & Human Services | NIH | National Institute on Alcohol Abuse and Alcoholism

U.S. Department of Health & Human Services | NIH | National Institute of Mental Health

Ministerio de Educación, Gobierno de Chile

Publisher

Springer Science and Business Media LLC

Subject

Psychiatry and Mental health,Pharmacology

Reference72 articles.

1. Murphy DL, Moya PR, Wendland JR, Timpano KR. Genetic contributions to obsessive-compulsive disorder (OCD) and OCD-related disorders. In: Berrettini JNW, (ed). Principles of psychiatric genetics. Cambridge, UK: Cambridge University Press; 2012. p. 121–33.

2. DSM-5. Diagnostic and statistical manual of mental disorders (DSM-5). 5th ed. Alrington, VA: American Psychiatric Association; 2013.

3. Koran LM, Hanna GL, Hollander E, Nestadt G, Simpson HB, American Psychiatric Association. Practice guideline for the treatment of patients with obsessive-compulsive disorder. Am J Psychiatry. 2007;164:5–53.

4. Ahmari SE, Dougherty DD. Dissecting OCD circuits: from animal models to targeted treatments. Depress Anxiety. 2015;32:550–62.

5. Pittenger C. Glutamatergic agents for OCD and related disorders. Curr Treat Options Psychiatry. 2015;2:271–83.

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