Mammalian APE1 controls miRNA processing and its interactome is linked to cancer RNA metabolism
Author:
Publisher
Springer Science and Business Media LLC
Subject
General Physics and Astronomy,General Biochemistry, Genetics and Molecular Biology,General Chemistry
Link
http://www.nature.com/articles/s41467-017-00842-8.pdf
Reference80 articles.
1. Li, M. & Wilson, D. M. Human apurinic/apyrimidinic endonuclease 1. Antioxid. Redox Signal. 20, 678–707 (2014).
2. Antoniali, G. et al. SIRT1 gene expression upon genotoxic damage is regulated by APE1 through nCaRE-promoter elements. Mol. Biol. Cell 25, 532–547 (2014).
3. Tell, G., Fantini, D. & Quadrifoglio, F. Understanding different functions of mammalian AP endonuclease (APE1) as a promising tool for cancer treatment. Cell Mol. Life Sci. 67, 3589–3608 (2010).
4. Chattopadhyay, R. et al. Regulatory role of human AP-endonuclease (APE1/Ref-1) in YB-1-mediated activation of the multidrug resistance gene MDR1. Mol. Cell Biol. 28, 7066–7080 (2008).
5. Sengupta, S., Mantha, A. K., Mitra, S. & Bhakat, K. K. Human AP endonuclease (APE1/Ref-1) and its acetylation regulate YB-1-p300 recruitment and RNA polymerase II loading in the drug-induced activation of multidrug resistance gene MDR1. Oncogene 30, 428–493 (2011).
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