Atypical fluorescence in situ hybridisation pattern in chronic myeloid leukaemia due to cryptic insertion of BCR at 9q34-Reference-Cited by-同舟云学术

Atypical fluorescence in situ hybridisation pattern in chronic myeloid leukaemia due to cryptic insertion of BCR at 9q34

Published:2003-11-06 Issue:1 Volume:18 Page:161-162
ISSN:0887-6924
Container-title:Leukemia
language:en
Short-container-title:Leukemia

Author:

Wan T S K,Ma S K,Li C K,Chan L C

Publisher

Springer Science and Business Media LLC

Subject

Oncology,Cancer Research,Hematology

Link

http://www.nature.com/articles/2403197.pdf

Reference8 articles.

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2. Huntly BJ, Reid AG, Bench AJ, Campbell LJ, Telford N, Shepherd P et al. Deletions of the derivative chromosome 9 occurs at the time of the Philadelphia translocation and provides a powerful and independent prognostic factor in chronic myeloid leukemia. Blood 2001; 98: 1732–1738.

3. Hagemeijer A, Buijs A, Smit E, Janssen B, Vreemers G-J, Van der Plas D et al. Translocation of BCR to chromosome 9: A new cytogenetic variant detected by FISH in two Ph-negative BCR-positive patients with chronic myeloid leukemia. Gene Chromosome Cancer 1993; 8: 237–245.

4. Dewald GW, Wyatt WA, Juneau AL, Carlson RO, Zinsmeister AR, Jalal SM et al. Highly sensitive fluorescence in situ hybridization method to detect double BCR/ABL fusion and monitor response to therapy in chronic myeloid leukemia. Blood 1998; 91: 3357–3365.

5. Sessarego M, Fugazza G, Bruzzone R, Ballestrero A, Miglino M, Bacigalupo A . Complex chromosome rearrangements may locate the bcr/abl fusion gene sites other than 22q11. Haematologica 2000; 85: 35–39.

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