Role for N-CoR and histone deacetylase in Sin3-mediated transcriptional repression

Author:

Alland Lelia,Muhle Rebecca,Hou Harry,Potes Jason,Chin Lynda,Schreiber-Agus Nicole,DePinho Ronald A.

Publisher

Springer Science and Business Media LLC

Subject

Multidisciplinary

Reference50 articles.

1. Henriksson, M. & Luscher, B. Proteins of the Myc network: essential regulators of cell growth and differentiation. Adv. Cancer Res. 68, 109–182 (1996).

2. Ayer, D. E., Lawrence, Q. A. & Eiseninan, R. N. Mad-Max transcriptional repression is mediated by ternary complex formation with mammalian homologs of yeast repressor Sin3. Cell 80, 767–776 (1995).

3. Schreiber-Agus, N. et al. An amino-terminal domain of Mxi1 mediates anti-Myc oncogenic activity and interacts with a homolog of the yeast transcriptional repressor SIN3. Cell 80, 777–786 (1995).

4. Koskinen, P. J., Ayer, D. E. & Eisenman, R. N. Repression of Myc-Ras cotransformation by Mad is mediated by multiple protein-protein interactions. Cell Growth Differ. 6, 623–629 (1995).

5. Rao, G. et al. Mouse Sin3A interacts with and can functionally substitute for the amino-terminal repression domain of the Myc antagonist Mxi. Oncogene 12, 1165–1172 (1996).

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