Cdc7-Dbf4-mediated phosphorylation of HSP90-S164 stabilizes HSP90-HCLK2-MRN complex to enhance ATR/ATM signaling that overcomes replication stress in cancer
Author:
Publisher
Springer Science and Business Media LLC
Subject
Multidisciplinary
Link
http://www.nature.com/articles/s41598-017-17126-2.pdf
Reference78 articles.
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3. Rendtlew Danielsen, J. M. et al. HCLK2 is required for activity of the DNA damage response kinase ATR. The Journal of biological chemistry 284, 4140–4147, https://doi.org/10.1074/jbc.M808174200 (2009).
4. Flynn, R. L. & Zou, L. ATR: a master conductor of cellular responses to DNA replication stress. Trends in biochemical sciences 36, 133–140, https://doi.org/10.1016/j.tibs.2010.09.005 (2011).
5. Houtgraaf, J. H., Versmissen, J. & van der Giessen, W. J. A concise review of DNA damage checkpoints and repair in mammalian cells. Cardiovascular Revascularization Medicine 7, 165–172, https://doi.org/10.1016/j.carrev.2006.02.002 (2006).
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