KH176 Safeguards Mitochondrial Diseased Cells from Redox Stress-Induced Cell Death by Interacting with the Thioredoxin System/Peroxiredoxin Enzyme Machinery
Author:
Publisher
Springer Science and Business Media LLC
Subject
Multidisciplinary
Link
http://www.nature.com/articles/s41598-018-24900-3.pdf
Reference53 articles.
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2. Aon, M. A. et al. Glutathione/thioredoxin systems modulate mitochondrial H2 O2 emission: An experimental-computational study. J. Gen. Physiol. 139, 479–491 (2012).
3. Bleier, L. et al. Generator-specific targets of mitochondrial reactive oxygen species. Free Radic. Biol. Med. 78, 1–10 (2015).
4. D’Autréaux, B. & Toledano, M. B. ROS as signalling molecules: mechanisms that generate specificity in ROS homeostasis. Nat. Rev. Mol. Cell Biol. 8, 813–824 (2007).
5. Winterbourn, C. C. Reconciling the chemistry and biology of reactive oxygen species. Nat. Chem. Biol. 4, 278–286 (2008).
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