Supplementation with a selective amino acid formula ameliorates muscular dystrophy in mdx mice
Author:
Funder
Fondazione Cariplo
Professional Dietetics
Associazione Centro Dino Ferrari
Publisher
Springer Science and Business Media LLC
Subject
Multidisciplinary
Link
http://www.nature.com/articles/s41598-018-32613-w.pdf
Reference82 articles.
1. Emery, A. E. The muscular dystrophies. Bmj 317, 991–995 (1998).
2. Davies, K. E. & Nowak, K. J. Molecular mechanisms of muscular dystrophies: old and new players. Nat Rev Mol Cell Biol 7, 762–773 (2006).
3. Grounds, M. D., Radley, H. G., Lynch, G. S., Nagaraju, K. & De Luca, A. Towards developing standard operating procedures for pre-clinical testing in the mdx mouse model of Duchenne muscular dystrophy. Neurobiology of disease 31, 1–19, https://doi.org/10.1016/j.nbd.2008.03.008 (2008).
4. Cerletti, M. et al. Dystrophic phenotype of canine X-linked muscular dystrophy is mitigated by adenovirus-mediated utrophin gene transfer. Gene therapy 10, 750–757, https://doi.org/10.1038/sj.gt.3301941 (2003).
5. Deconinck, A. E. et al. Utrophin-dystrophin-deficient mice as a model for Duchenne muscular dystrophy. Cell 90, 717–727 (1997).
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