Genetic and animal model analyses reveal the pathogenic role of a novel deletion of RELN in schizophrenia
Author:
Publisher
Springer Science and Business Media LLC
Subject
Multidisciplinary
Link
http://www.nature.com/articles/s41598-018-31390-w.pdf
Reference58 articles.
1. Ogawa, M. et al. The Reeler Gene-Associated Antigen on Cajal-Retzius Neurons Is a Crucial Molecule for Laminar Organization of Cortical-Neurons. Neuron 14, 899–912, https://doi.org/10.1016/0896-6273(95)90329-1 (1995).
2. Tissir, F. & Goffinet, A. M. Reelin and brain development. Nat Rev Neurosci 4, 496–505, https://doi.org/10.1038/nrn1113 (2003).
3. Arioka, Y. et al. Single-cell trajectory analysis of human 1 homogenous neurons carrying a rare RELN variant. Transl Psychiatry (2018).
4. Qui, S. F. et al. Cognitive disruption and altered hippocampus synaptic function in Reelin haploinsufficient mice. Neurobiol Learn Mem 85, 228–242, https://doi.org/10.1016/j.nlm.2005.11.001 (2006).
5. Herz, J. & Chen, Y. Reelin, lipoprotein receptors and synaptic plasticity. Nat Rev Neurosci 7, 850–859, https://doi.org/10.1038/nrn2009 (2006).
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