Improvement of vascular dysfunction by argirein through inhibiting endothelial cell apoptosis associated with ET-1/Nox4 signal pathway in diabetic rats-Reference-Cited by-同舟云学术

Improvement of vascular dysfunction by argirein through inhibiting endothelial cell apoptosis associated with ET-1/Nox4 signal pathway in diabetic rats

Published:2018-08-22 Issue:1 Volume:8 Page:
ISSN:2045-2322
Container-title:Scientific Reports
language:en
Short-container-title:Sci Rep

Author:

Su Jie,An Xing-Rong,Li Qing,Li Xiao-Xue,Cong Xiao-dong,Xu Ming

Publisher

Springer Science and Business Media LLC

Subject

Multidisciplinary

Link

http://www.nature.com/articles/s41598-018-30386-w.pdf

Reference39 articles.

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2. Prakash, K., Chandran, D. S., Khadgawat, R., Jaryal, A. K. & Deepak, K. K. Correlations between endothelial function in the systemic and cerebral circulation and insulin resistance in type 2 diabetes mellitus. Diabetes & vascular disease research 13, 49–55, https://doi.org/10.1177/1479164115604120 (2016).

3. Wang, Q. et al. Activation of NAD(P)H oxidase by tryptophan-derived 3-hydroxykynurenine accelerates endothelial apoptosis and dysfunction in vivo. Circ Res 114, 480–492, https://doi.org/10.1161/CIRCRESAHA.114.302113 (2014).

4. Cong, X. D., Fu, P. R., Dai, D. Z., Zhang, Y. & Dai, Y. Pharmacokinetic behavior of argirein, derived from rhein, is characterized as slow release and prolonged T(1)/(2) of rhein in rats. European journal of pharmaceutical sciences: official journal of the European Federation for Pharmaceutical Sciences 46, 468–474, https://doi.org/10.1016/j.ejps.2012.03.014 (2012).

5. Shi, F. H. et al. Depressed calcium-handling proteins due to endoplasmic reticulum stress and apoptosis in the diabetic heart are attenuated by argirein. Naunyn-Schmiedeberg’s archives of pharmacology 386, 521–531, https://doi.org/10.1007/s00210-013-0852-5 (2013).

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