The Expression of Toll-like receptors in eutopic and ectopic endometrium and its implication in the inflammatory pathogenesis of adenomyosis
Author:
Publisher
Springer Science and Business Media LLC
Subject
Multidisciplinary
Link
http://www.nature.com/articles/s41598-017-07859-5.pdf
Reference19 articles.
1. Struble, J., Reid, S. & Bedaiwy, M. A. Adenomyosis: A Clinical Review of a Challenging Gynecologic Condition. J Minim Invasive Gynecol 23, 164–185 (2016).
2. Carrarelli, P. et al. Expression of Inflammatory and Neurogenic Mediators in Adenomyosis: A Pathogenetic Role. Reprod Sci (2016).
3. Scheerer, C. et al. Characterization of endometriosis-associated immune cell infiltrates (EMaICI). Arch Gynecol Obstet 294, 657–664 (2016).
4. Khan, K. N. et al. Additive effects of inflammation and stress reaction on Toll-like receptor 4-mediated growth of endometriotic stromal cells. Hum Reprod 28, 2794–2803 (2013).
5. Banu, S. K., Lee, J. Jr., Speights, V. O., Starzinski-Powitz, A. & Arosh, J. A. Cyclooxygnemase-2 regulates survival, migration, and invasion of human endometriotic cells through multiple mechanisms. Endocrinology 149, 1180–1189 (2008).
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