Burn-induced muscle metabolic derangements and mitochondrial dysfunction are associated with activation of HIF-1α and mTORC1: Role of protein farnesylation-Reference-Cited by-同舟云学术

Burn-induced muscle metabolic derangements and mitochondrial dysfunction are associated with activation of HIF-1α and mTORC1: Role of protein farnesylation

Published:2017-07-26 Issue:1 Volume:7 Page:
ISSN:2045-2322
Container-title:Scientific Reports
language:en
Short-container-title:Sci Rep

Author:

Nakazawa Harumasa,Ikeda Kazuhiro,Shinozaki Shohei,Kobayashi Masayuki,Ikegami Yuichi,Fu Ming,Nakamura Tomoyuki,Yasuhara Shingo,Yu Yong-Ming,Martyn J. A. Jeevendra,Tompkins Ronald G.,Shimokado Kentaro,Yorozu Tomoko,Ito Hideki,Inoue Satoshi,Kaneki Masao

Publisher

Springer Science and Business Media LLC

Subject

Multidisciplinary

Link

http://www.nature.com/articles/s41598-017-07011-3.pdf

Reference67 articles.

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2. Ogunbileje, J. O. et al. Hypermetabolism and hypercatabolism of skeletal muscle accompany mitochondrial stress following severe burn trauma. American journal of physiology. Endocrinology and metabolism 311, E436–448 (2016).

3. Porter et al. The impact of severe burns on skeletal muscle mitochondrial function. Burns 39, 1039–1047 (2013).

4. Ueki, R. et al. Role of Elevated Fibrinogen in Burn-Induced Mitochondrial Dysfunction: Protective Effects of Glycyrrhizin. Shock (Augusta, Ga.) 46, 382–389 (2016).

5. Cree, M. et al. Role of fat metabolism in burn trauma-induced skeletal muscle insulin resistance. Crit Care Med 35, S476–483 (2007).

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