Activation of Endoplasmic Reticulum Stress in Granulosa Cells from Patients with Polycystic Ovary Syndrome Contributes to Ovarian Fibrosis-Reference-Cited by-同舟云学术

Activation of Endoplasmic Reticulum Stress in Granulosa Cells from Patients with Polycystic Ovary Syndrome Contributes to Ovarian Fibrosis

Published:2017-09-07 Issue:1 Volume:7 Page:
ISSN:2045-2322
Container-title:Scientific Reports
language:en
Short-container-title:Sci Rep

Author:

Takahashi Nozomi,Harada Miyuki,Hirota Yasushi^ORCID,Nose Emi,Azhary Jerilee MK,Koike Hiroshi,Kunitomi Chisato,Yoshino Osamu,Izumi Gentaro,Hirata Tetsuya,Koga Kaori,Wada-Hiraike Osamu,Chang R. Jeffrey,Shimasaki Shunichi,Fujii Tomoyuki,Osuga Yutaka

Publisher

Springer Science and Business Media LLC

Subject

Multidisciplinary

Link

http://www.nature.com/articles/s41598-017-11252-7.pdf

Reference59 articles.

1. Bozdag, G., Mumusoglu, S., Zengin, D., Karabulut, E. & Yildiz, B. O. The prevalence and phenotypic features of polycystic ovary syndrome: a systematic review and meta-analysis. Hum Reprod. 31, 2841–2855 (2016).

2. Dumesic, D. A. et al. Scientific Statement on the Diagnostic Criteria, Epidemiology, Pathophysiology, and Molecular Genetics of Polycystic Ovary Syndrome. Endocr Rev. 36, 487–525 (2015).

3. Ma, X. et al. Proteomic analysis of human ovaries from normal and polycystic ovarian syndrome. Mol Hum Reprod. 13, 527–535 (2007).

4. Schmidt, J. et al. Differential expression of inflammation-related genes in the ovarian stroma and granulosa cells of PCOS women. Molr Human Reprod. 20, 49–58 (2014).

5. Zhao, Y. et al. Up-regulated expression of WNT5a increases inflammation and oxidative stress via PI3K/AKT/NF-kappaB signaling in the granulosa cells of PCOS patients. J Clin Endocrinol Metab. 100, 201–211 (2015).

Cited by 89 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. BRD4 absence inactivates endoplasmic reticulum stress to retard dehydroepiandrosterone-triggered ovarian granular cell apoptosis in polycystic ovary syndrome via GRP78;Tissue and Cell;2024-12

2. Endoplasmic Reticulum Stress in Myometrial Smooth Muscle Cells and Spontaneous Contraction Changes in the Uterus of Dehydroepiandrosterone-induced Polycystic Ovary Syndrome Rats;Cell Biochemistry and Biophysics;2024-09-11

3. Endoplasmic reticulum stress-mediated ferroptosis in granulosa cells contributes to follicular dysfunction of polycystic ovary syndrome driven by hyperandrogenism;Reproductive BioMedicine Online;2024-09

4. Mixtures of phthalates disrupt expression of genes related to lipid metabolism and peroxisome proliferator-activated receptor signaling in mouse granulosa cells;Toxicological Sciences;2024-08-16

5. IL-18BP Therapy Ameliorates Reproductive and Metabolic Phenotypes in a PCOS Mouse Model by Relieving Inflammation, Fibrosis and Endoplasmic Reticulum Stress;Reproductive Sciences;2024-07-08