Lactate oxidation facilitates growth of Mycobacterium tuberculosis in human macrophages
Author:
Publisher
Springer Science and Business Media LLC
Subject
Multidisciplinary
Link
http://www.nature.com/articles/s41598-017-05916-7.pdf
Reference52 articles.
1. Zumla, A. et al. The WHO 2014 global tuberculosis report–further to go. Lancet Glob. Health. 3, e10–2 (2015).
2. Marrero, J., Rhee, K. Y., Schnappinger, D., Pethe, K. & Ehrt, S. Gluconeogenic carbon flow of tricarboxylic acid cycle intermediates is critical for Mycobacterium tuberculosis to establish and maintain infection. Proc. Natl. Acad. Sci. USA 107, 9819–9824 (2010).
3. Bloch, H. & Segal, W. Biochemical differentiation of Mycobacterium tuberculosis grown in vivo and in vitro. J. Bacteriol. 72, 132–141 (1956).
4. McKinney, J. D. et al. Persistence of Mycobacterium tuberculosis in macrophages and mice requires the glyoxylate shunt enzyme isocitrate lyase. Nature 406, 735–738 (2000).
5. Schnappinger, D. et al. Transcriptional Adaptation of Mycobacterium tuberculosis within Macrophages: Insights into the Phagosomal Environment. J. Exp. Med. 198, 693–704 (2003).
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