The Role of Inflammation in β-cell Dedifferentiation-Reference-Cited by-同舟云学术

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The Role of Inflammation in β-cell Dedifferentiation

Published:2017-07-24 Issue:1 Volume:7 Page:
ISSN:2045-2322
Container-title:Scientific Reports
language:en
Short-container-title:Sci Rep

Author:

Nordmann Thierry M.,Dror Erez,Schulze Friederike,Traub Shuyang,Berishvili Ekaterine,Barbieux Charlotte,Böni-Schnetzler Marianne,Donath Marc Y.

Publisher

Springer Science and Business Media LLC

Subject

Multidisciplinary

Link

http://www.nature.com/articles/s41598-017-06731-w.pdf

Reference29 articles.

1. Tabak, A. G. et al. Trajectories of glycaemia, insulin sensitivity, and insulin secretion before diagnosis of type 2 diabetes: an analysis from the Whitehall II study. Lancet 373, 2215–2221, doi: 10.1016/S0140-6736(09)60619-X (2009).

2. Boni-Schnetzler, M. et al. Free fatty acids induce a proinflammatory response in islets via the abundantly expressed interleukin-1 receptor I. Endocrinology 150, 5218–5229, doi: 10.1210/en.2009-0543 (2009).

3. Back, S. H. & Kaufman, R. J. Endoplasmic reticulum stress and type 2 diabetes. Annu Rev Biochem 81, 767–793, doi: 10.1146/annurev-biochem-072909-095555 (2012).

4. Masters, S. L. et al. Activation of the NLRP3 inflammasome by islet amyloid polypeptide provides a mechanism for enhanced IL-1beta in type 2 diabetes. Nat Immunol 11, 897–904, doi: 10.1038/ni.1935 (2010).

5. Westwell-Roper, C. Y., Ehses, J. A. & Verchere, C. B. Resident macrophages mediate islet amyloid polypeptide-induced islet IL-1beta production and beta-cell dysfunction. Diabetes 63, 1698–1711, doi: 10.2337/db13-0863 (2014).

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