LGR4 is a receptor for RANKL and negatively regulates osteoclast differentiation and bone resorption-Reference-Cited by-同舟云学术

LGR4 is a receptor for RANKL and negatively regulates osteoclast differentiation and bone resorption

Published:2016-04-11 Issue:5 Volume:22 Page:539-546
ISSN:1078-8956
Container-title:Nature Medicine
language:en
Short-container-title:Nat Med

Author:

Luo Jian^ORCID,Yang Zhengfeng^ORCID,Ma Yu,Yue Zhiying,Lin Hongyu,Qu Guojun,Huang Jinping,Dai Wentao,Li Chenghai,Zheng Chunbing,Xu Leqin,Chen Huaqing,Wang Jiqiu,Li Dali,Siwko Stefan,Penninger Josef M^ORCID,Ning Guang,Xiao Jianru,Liu Mingyao

Publisher

Springer Science and Business Media LLC

Subject

General Biochemistry, Genetics and Molecular Biology,General Medicine

Link

http://www.nature.com/articles/nm.4076.pdf

Reference44 articles.

1. Kong, Y.Y. et al. OPGL is a key regulator of osteoclastogenesis, lymphocyte development and lymph-node organogenesis. Nature 397, 315–323 (1999).

2. Hanada, R., Hanada, T., Sigl, V., Schramek, D. & Penninger, J.M. RANKL/RANK-beyond bones. J. Mol. Med. 89, 647–656 (2011).

3. Fata, J.E. et al. The osteoclast differentiation factor osteoprotegerin-ligand is essential for mammary gland development. Cell 103, 41–50 (2000).

4. Jones, D.H. et al. Regulation of cancer cell migration and bone metastasis by RANKL. Nature 440, 692–696 (2006).

5. Gonzalez-Suarez, E. et al. RANK ligand mediates progestin-induced mammary epithelial proliferation and carcinogenesis. Nature 468, 103–107 (2010).

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