Direct interaction between CD155 and CD96 promotes immunosuppression in lung adenocarcinoma-Reference-Cited by-同舟云学术

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Direct interaction between CD155 and CD96 promotes immunosuppression in lung adenocarcinoma

Published:2020-09-11 Issue:6 Volume:18 Page:1575-1577
ISSN:1672-7681
Container-title:Cellular & Molecular Immunology
language:en
Short-container-title:Cell Mol Immunol

Author:

Zhang Hui,Liu Qianwen,Lei Yiyan,Zhou Jianwen,Jiang Wenting,Cui Yongmei,He Qiong,Zhu Junfeng,Zhu Zheng,Sun Yu,Ke Zunfu

Publisher

Springer Science and Business Media LLC

Subject

Infectious Diseases,Immunology,Immunology and Allergy

Link

https://www.nature.com/articles/s41423-020-00538-y.pdf

Reference8 articles.

1. Shibuya, A. et al. DNAM-1, a novel adhesion molecule involved in the cytolytic function of T lymphocytes. Immunity 4, 573–581 (1996).

2. Bottino, C. et al. Identification of PVR (CD155) and Nectin-2 (CD112) as cell surface ligands for the human DNAM-1 (CD226) activating molecule. J. Exp. Med. 198, 557–567 (2003).

3. Yu, X. et al. The surface protein TIGIT suppresses T cell activation by promoting the generation of mature immunoregulatory dendritic cells. Nat. Immunol. 10, 48–57 (2009).

4. Iguchi-Manaka, A. et al. Increased soluble CD155 in the serum of cancer patients. PloS ONE 11, e0152982 (2016).

5. Brooks, J. et al. Perioperative, spatiotemporally coordinated activation of T and NK cells prevents recurrence of pancreatic cancer. Cancer Res. 78, 475–488 (2017).

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