Dual inhibition of Jak2 and STAT5 enhances killing of myeloproliferative neoplasia cells
Author:
Publisher
Springer Science and Business Media LLC
Subject
Oncology,Cancer Research,Hematology
Link
http://www.nature.com/articles/leu2011338.pdf
Reference14 articles.
1. Levine RL, Gilliland DG . Myeloproliferative disorders. Blood 2008; 112: 2190–2198.
2. Delhommeau F, Pisani DF, James C, Casadevall N, Constantinescu S, Vainchenker W . Oncogenic mechanisms in myeloproliferative disorders. Cell Mol Life Sci 2006; 63: 2939–2953.
3. Wernig G, Mercher T, Okabe R, Levine RL, Lee BH, Gilliland DG . Expression of Jak2V617F causes a polycythemia vera-like disease with associated myelofibrosis in a murine bone marrow transplant model. Blood 2006; 107: 4274–4281.
4. Pardanani A, Hood J, Lasho T, Levine RL, Martin MB, Noronha G et al. TG101209, a small molecule JAK2-selective kinase inhibitor potently inhibits myeloproliferative disorder-associated JAK2V617F and MPLW515L/K mutations. Leukemia 2007; 21: 1658–1668.
5. Goldman JM, Green AR, Holyoake T, Jamieson C, Mesa R, Mughal T et al. Chronic myeloproliferative diseases with and without the Ph chromosome: some unresolved issues. Leukemia 2009; 23: 1708–1715.
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